[1] Larsen FS, Wendon J. Understanding paracetamol-induced liver failure. Intensive Care Med, 2014, 40:888-890. [2] Rahman TM, Selden AC, Hodgson HJ. A novel model of acetaminophen-induced acute hepatic failure in rabbits. J Surg Res, 2002, 106:264-272. [3] Kelly JH, Koussayer T, He DE, et al. An improved model of acetaminophen-induced fulminant hepatic failure in dogs. Hepatology, 1992, 15:329-335. [4] Francavilla A, Makowka L, Polimeno L, et al. A dog model for acetaminophen-induced fulminant hepatic failure. Gastroenterology, 1989, 96:470-478. [5] Rumbeiha WK, Lin YS, Oehme FW. Comparison of N-acetylcysteine and methylene blue, alone or in combination, for treatment of acetaminophen toxicosis in cats. Am J Vet Res, 1995, 56:1529-1533. [6] Liu P, McGuire GM, Fisher MA, et al. Activation of Kupffer cells and neutrophils for reactive oxygen formation is responsible for endotoxin-enhanced liver injury after hepatic ischemia. Shock, 1995, 3:56-62. [7] Laskin DL, Gardner CR, Price VF, et al. Modulation of macrophage functioning abrogates the acute hepatotoxicity of acetaminophen. Hepatology, 1995, 21:1045-1050. [8] Kofman AV, Morgan G, Kirschenbaum A, et al. Dose- and time-dependent oval cell reaction in acetaminophen-induced murine liver injury. Hepatology, 2005, 41:1252-1261. [9] Ramachandran A, McGill MR, Xie Y, et al. Receptor interacting protein kinase 3 is a critical early mediator of acetaminophen-induced hepatocyte necrosis in mice. Hepatology, 2013, 58:2099-2108. [10] 李继强. 急性肝衰竭的病理表现. 肝脏, 1997, 2:93-94. [11] Numata K, Kubo M, Watanabe H, et al. Overexpression of suppressor of cytokine signaling-3 in T cells exacerbates acetaminophen-induced hepatotoxicity. J Immunol, 2007, 178:3777-3785. [12] Yang R, Zhang S, Cotoia A, et al. High mobility group B1 impairs hepatocyte regeneration in acetaminophen hepatotoxicity. BMC Gastroenterol, 2012, 12:45. [13] Zaher H, Buters JT, Ward JM, et al. Protection against acetaminophen toxicity in CYP1A2 and CYP2E1 double-null mice. Toxicol Appl Pharmacol, 1998, 152:193-199. [14] Mitchell JR, Jollow DJ, Potter WZ, et al. Acetaminophen-induced hepatic necrosis. IV. Protective role of glutathione. J Pharmacol Exp Ther, 1973, 187:211-217. [15] Davis DC, Potter WZ, Jollow DJ, et al. Species differences in hepatic glutathione depletion, covalent binding and hepatic necrosis after acetaminophen. Life Sci, 1974, 14:2099-2109. [16] McGill MR, Williams CD, Xie Y, et al. Acetaminophen-induced liver injury in rats and mice: comparison of protein adducts, mitochondrial dysfunction, and oxidative stress in the mechanism of toxicity. Toxicol Appl Pharmacol, 2012, 264:387-394. [17] McGill MR, Jaeschke H. Metabolism and disposition of acetaminophen: recent advances in relation to hepatotoxicity and diagnosis. Pharm Res, 2013, 30:2174-2187. [18] Saracyn M, Zdanowski R, Brytan M, et al. D-Galactosamine Intoxication in Experimental Animals: Is it Only an Experimental Model of Acute Liver Failure? Med Sci Monit, 2015, 21:1469-1477. [19] Hanawa N, Shinohara M, Saberi B, et al. Role of JNK translocation to mitochondria leading to inhibition of mitochondria bioenergetics in acetaminophen-induced liver injury. J Biol Chem, 2008, 283:13565-13577. [20] Kon K, Kim JS, Jaeschke H, et al. Mitochondrial permeability transition in acetaminophen-induced necrosis and apoptosis of cultured mouse hepatocytes. Hepatology, 2004, 40:1170-1179. |