[1] Li L, Wang H. Heterogeneity of liver cancer and personalized therapy.. Cancer Letters, 2016, 379:191-197. [2] Wan S , Meyer AS , Weiler SME . Cytoplasmic localization of the cell polarity factor Scribble supports liver tumor formation and tumor cell invasiveness.. Hepatology, 2018, 67:1842-1856. [3] Scott M,Hyland PL,Mcgregor G,et al.Multimodality expression profiling shows SEPT 9 to be overexpressed in a wide range of human tumours. Oncogene,2005,24:4688-4700. [4] Oussalah A, Rischer S, Bensenane M, et al. Plasma mSEPT9: A Novel Circulating Cell-free DNA-Based Epigenetic Biomarker to Diagnose Hepatocellular Carcinoma. Ebiomedicine, 2018,4:138-142. [5] Bai X , Karasmanis EP , Spiliotis ET . Septin 9 interacts with kinesin KIF17 and interferes with the mechanism of NMDA receptor cargo binding and transport. Mol Biol Cell, 2016, 27:897-906. [6] Reed S O , Lamason R , Risca V , et al. Rickettsia Actin-Based Motility Occurs in Distinct Phases Mediated by Different Actin Nucleators. Curr Biol, 2014, 24:98-103. [7] Song L,Yu H,Jia J,et al. A systematic review of the performance of the SEPT 9 gene methylation assay in colorectal cancer screening,monitoring,diagnosis and prognosis. Cancer Biomark, 2017,18:425-432. [8] 刘文, 狄建忠, 邹扬,等. SEPT9基因与恶性肿瘤关系的研究进展. 肿瘤, 2010, 30:798-801. [9] Flomerfelt FA, Gress RE. Analysis of Cell Proliferation and Homeostasis Using EdU Labeling. Methods Mol Biol, 2016, 1323:211-216. [10] Carvalho MI, Pires I, Prada J, et al. Ki-67 and PCNA Expression in Canine Mammary Tumors and Adjacent Nonneoplastic Mammary Glands: Prognostic Impact by a Multivariate Survival Analysis. Vet Pathol, 2016, 53:1138-1146. [11] Wang Y, Chen T, Huang H, et al. miR-363-3p inhibits tumor growth by targeting PCNA in lung adenocarcinoma. Oncotarget, 2017, 8:20133-20144. [12] 童宇洋, 许晓琳, 罗葆明. 上皮-间质转化在肝癌非手术治疗后促进残癌侵袭、转移的研究进展. 中国介入影像与治疗学, 2016, 13:449-452. [13] Li FJ, Surolia R, Li H, et al. Autoimmunity to Vimentin Is Associated with Outcomes of Patients with Idiopathic Pulmonary Fibrosis.. J Immunol, 2017, 199:1596-1605. [14] Labernadie A, Kato T, Brugués A, et al. A mechanically active heterotypic E-cadherin/N-cadherin adhesion enables fibroblasts to drive cancer cell invasion. Nat Cell Biol, 2017, 19:224-231. [15] Chang YC , Chan YC , Chang WM , et al. Feedback regulation of ALDOA activates the HIF-1α/MMP9 axis to promote lung cancer progression. Cancer Lett, 2017, 10:28-36. [16] Jablonska-Trypuć A, Matejczyk M,et al. Matrix metalloproteinases (MMPs), the main extracellular matrix (ECM) enzymes in collagen degradation, as a target for anticancer drugs. J Enzyme Inhib Med Chem, 2016, 23:177-183. |