[1] Walewska A, Szewczyk A, Koprowski P. Gas signaling molecules and mitochondrial potassium channels.Int J Mol Sci,2018,19:3227.
[2] Schatzschneider U. Novel lead structures and activation mechanisms for CO- releasing molecules (CORMs). Br J Pharmacol. 2015, 172 :1638-1650.
[3] Marhenke J, Trevino K, Works C. The chemistry, biology and design of photochemical CO releasing molecules and the efforts to detect CO for biological applications. Coord Chem Rev, 2016, 306:533-543.
[4] Ling K, Men F, Wang WC, et al. Carbon monoxide and its controlled release: therapeutic application, detection, and development of carbon monoxide releasing molecules (CORMs). J Med Chem, 2018 , 61:2611-2635.
[5] Faizan M, Muhammad N, Niazi KUK, et al. CO-releasing materials: an emphasis on therapeutic implications, as release and subsequent cytotoxicity are the part of therapy. Materials (Basel), 2019, 12:1643.
[6] Ismailova A?, Kuter D?, Bohle DS, et al. An overview of the potential therapeutic applications of CO-Releasing molecules. Bioinorg Chem Appl, 2018,2018:8547364.
[7] Seveso M, Vadori M, Bosio E, et al. Pharmacological effects and tolerability profile of a carbon monoxide-releasing molecule (CORM-3) in primates. Am J Transplantation, 2005,5:306-307.
[8] Wang P, Liu H, Zhao Q, et al. Syntheses and evaluation of drug-like properties of CO-releasing molecules containing ruthenium and group 6 metal. Eur J Med Chem, 2014, 74:199-215.
[9] Seixas JD, Santos MF, Mukhopadhyay A, et al. A contribution to the rational design of Ru(CO)3Cl2L complexes for in vivo delivery of CO. Dalton Trans,2015,44: 5058-5075.
[10] Feger M, Fajol A, Lebedeva A, et al. Effect of carbon monoxide donor CORM-2 on vitamin D3 metabolism. Kidney Blood Press Res, 2013, 37: 496-505.
[11] Winburn IC, Gunatunga K, McKernan RD, et al. Cell damage following carbon monoxide releasing molecule exposure: implications for therapeutic applications. Basic Clin Pharmacol Toxicol, 2012,111: 31-41.
[12] Sun J, Guo E, Yang J, et al. Carbon monoxide ameliorates hepatic ischemia/ reperfusion injury via sirtuin 1-mediated deacetylation of high-mobility group box 1 in rats. Liver Transpl, 2017, 23:510-526.
[13] Pizarro MD, Rodriguez JV, Mamprin ME, et al. Protective effects of a carbon monoxide-releasing molecule (CORM-3) during hepatic cold preservation. Cryobiology,2009 ;58:248-255.
[14] Murphy PB, Bihari A, Parry NG, et al. Carbon monoxide and hydrogen sulphide reduce reperfusion injury in abdominal compartment syndrome. J Surg Res, 2018, 222:17-25.
[15] Wei Y, Chen P, de Bruyn M, et al. Carbon monoxide-releasing molecule-2 (CORM-2) attenuates acute hepatic ischemia reperfusion injury in rats. BMC Gastroenterol, 2010,10:42.
[16] 何大立,张玄,慕喜喜,等.低浓度CO暴露促进大鼠肝前体细胞增殖的体外研究.现代生物医学进展,2017,17:1601-1605.
[17] Wang X, Qin W, Song M, et al. Exogenous carbon monoxide inhibits neutrophil infiltration in LPS-induced sepsis by interfering with FPR1 via p38 MAPK but not GRK2.Oncotarget, 2016,7:34250-34265.
[18] Shen WC, Wang X, Qin WT, et al. Exogenous carbon monoxide suppresses Escherichia coli vitality and improves survival in an Escherichia coli-induced murine sepsis model. Acta Pharmacol Sin, 2014,35:1566-1576.
[19] Liang F, Cao J, Qin WT, et al. Regulatory effect and mechanisms of carbon monoxide-releasing molecule II on hepatic energy metabolism in septic mice. World J Gastroenterol,2014, 20:3301-3311.
[20] Cepinskas G, Katada K, Bihari A. Carbon monoxide liberated from carbon monoxide-releasing molecule CORM-2 attenuates inflammation in the liver of septic mice. Am J Physiol Gastrointest Liver Physiol,2008, 294:184-191.
[21] 石庚生. 外源性一氧化碳释放分子(CORM-2)对脓毒症早期肝脏炎症反应和JAK分子磷酸化的抑制作用及分子机制.江苏大学,2010.
[22] Upadhyay KK, Jadeja RN, Thadani JM, et al. Carbon monoxide releasing molecule A-1 attenuates acetaminophen-mediated hepatotoxicity and improves survival of mice by induction of Nrf2 and related genes. Toxicol Appl Pharmacol, 2018, 360:99-108.
[23] Yan BZ, Yang BS ,Li H, et al. The therapeutic effect of CORM-3 on acute liver failure induced by lipopolysaccharide/D-galactosamine in mice. Hepatobiliary Pancreat Dis Int, 2016,15:73-80.
[24] 颜炳柱,王晓韧,许磊,等.一氧化碳释放分子-2对小鼠急性肝功能衰竭保护作用的实验研究.肝脏,2015,8:602-605.
[25] Bakhautdin B, Das D, Mandal P, et al. Protective role of HO-1 and carbon monoxide in ethanol-induced hepatocyte cell death and liver injury in mice. J Hepatol, 2014,61:1029-1037.
[26] Li Y, Gao C, Shi Y, et al. Carbon monoxide alleviates ethanol-induced oxidative damage and inflammatory stress through activating p38 MAPK pathway. Toxicol Appl Pharmacol, 2013,273:53-58.
[27] Kim HJ, Joe Y, Kim SK, et al. Carbon monoxide protects against hepatic steatosis in mice by inducing sestrin-2 via the PERK-eIF2α-ATF4 pathway. Free Radic Biol Med,2017,110:81-91.
[28] Mangano K, Cavalli E, Mammana S, et al. Involvement of the Nrf2/HO-1/CO axis and therapeutic intervention with the CO-releasing molecule CORM-A1, in a murine model of autoimmune hepatitis.J Cell Physiol,2018,233:4156-4165.
[29] Zhang L, Zhang Z, Liu B, et al. The protective effect of heme oxygenase-1 against intestinal barrier dysfunction in cholestatic liver injury is associated with NF-κB inhibition. Mol Med, 2017, 23:215-224.
[30] Van Landeghem L, Laleman W, Vander Elst I, et al. Carbon monoxide produced by intrasinusoidally located haem-oxygenase-1 regulates the vascular tone in cirrhotic rat liver. Liver Int, 2009,29:650-660. |
