[1] Rodriguez Roisin R,Krowka M J.Hepatopulmonary syndrome a liver-induced lung vascular disorder.N Engl J Med,2008,358:2378-2387. [2] Soulaidopoulos S,Cholongitas E,Giannakoulas G,et al.Review article:Update on current and emergent data on hepatopulmonary syndrome.World J Gastroenterol, 2018,24:1285-1298. [3] 李宁,郝建宇,庞宝森,等.高压氧治疗肝肺综合征大鼠的实验研究.肝脏, 2007, 12:103-107. [4] Zhang J,Yang W,Luo B,et al.The role of CX3 CL1/CX3CR1 in pulmonary angiogene-sis and intravascular monocyte accumulation in rat experimental hepatopulmonary syndrome.Hepatology,2012,57:752-758. [5] Kishimoto T.IL-6:from its discovery to clinical applications.Int lmmunol,2010,22:347-352. [6] 诸海军,万健,孙莲娜,等.WBC、IL-6及NLR值对慢加急性肝衰竭患者的早期预警价值.肝脏,2019,24:1076-1078. [7] Zhang J,Luo B,Tang L,et,al.Pulmonary angiogenesis in a rat model of hepatopu-lmonary syndrome.Hepatobiliary Pancreat Dis Int Gastroenterology,2009,136:1070-1080. [8] Garcia-ayllon MS,Silveyra MX,Candela A,et al.Changes in liver and plasma acetylcholinesterase in rats with cirrhosis induced by bile duct ligation. Hepatology,2006,43:444-453. [9] Raevens,Geerts A,Van Steenkiste C,et al.Hepatopulmonary syndrome and porto-pulmonary hypertension:recent knowledge in pathogenesis and overview of clinical assessment.Liver Int,2015,35:1646-1660. [10] Raevens S,Fallon MB.Potential Clinical targets in hepatopulmonary syndrome:Le-ssons from experimental models.Hepatology,2018,68:2016-2028. [11] Palma DT,Fallon MB. The hepatopulmonary syndrome. Hepatology,2006, 45:617-625. [12] Rose-John S.IL-6 trans-signaling via the soluble IL-6 receptor:importance for the pro-inflammatory activities of IL-6.Int J Biol Sci,2012,8:1237-1247. [13] Garbers C,Aparicio-Siegmund S,Rose-John S.The IL-6/gp130/STAT3 signaling axis:recent advances towards specific inhibition.Curr Opin Immunol, 2015,34:75-82. [14] Baratta M G.STAT3 true colours.Nat Rev Cancer,2018,18:664-665. |