[1] Lauer GM, Walker BD. Hepatitis C virus infection. New Engl J Med,2001, 345: 41-52.
[2] Moradpour D, Penin F, Rice CM. Replication of hepatitis C virus. Nat Rev Microbiol,2007, 5: 453-463.
[3] Shulla A, Randall G. Hepatitis C virus-host interactions, replication, and viral assembly. Curr Opin Virol,2012, 2: 725-732.
[4] Pasquinelli AE. MicroRNAs and their targets: recognition, regulation and an emerging reciprocal relationship. Nat Rev Genet,2012, 13: 271-282.
[5] Croce CM. Causes and consequences of microRNA dysregulation in cancer. Nat Rev Genet,2009, 10: 704-714.
[6] tenOever BR. RNA viruses and the host microRNA machinery. Nat Rev Microbiol,2013, 11: 169-180.
[7] Szabo G, Bala S. MicroRNAs in liver disease. Nat Rev Gastroenterol Hepatol, 2013, 10: 542-552.
[8] Jopling CL, Yi MK, Lancaster AM, et al. Modulation of hepatitis C virus RNA abundance by a liver-specific MicroRNA. Science,2005, 309: 1577-1581.
[9] Li Y, Masaki T, Yamane D, et al. Competing and noncompeting activities of miR-122 and the 5′ exonuclease Xrn1 in regulation of hepatitis C virus replication. Proc Natl Acad Sci USA, 2013, 110: 1881-1886.
[10] Conrad KD, Niepmann M. The role of microRNAs in hepatitis C virus RNA replication. Arch Virol, 2014, 159: 849-862.
[11] Pedersen IM, Cheng G, Wieland S, et al. Interferon modulation of cellular microRNAs as an antiviral mechanism. Nature,2007, 449: 919-922.
[12] Mukherjee A, Shrivastava S, Chowdhury JB, et al. Transcriptional suppression of miR-181c by hepatitis C virus enhances homeobox A1 expression. J Virol, 2014, 88: 7929-7940.
[13] Heim MH. Innate immunity and HCV. J Hepatol, 2013, 58: 564-574.
[14] Chowdhury JB, Shrivastava S, Steele R, et al. Hepatitis C virus infection modulates expression of interferon stimulatory gene IFITM1 by upregulating miR-130A. J Virol, 2012, 86: 10221-10225.
[15] Li S, Duan X, Li Y, et al. MicroRNA-130a inhibits HCV replication by restoring the innate immune response. J Viral Hepat, 2014, 21: 121-128.
[16] Chen Y, Chen J, Wang H, et al. HCV-induced miR-21 contributes to evasion of host immune system by targeting MyD88 and IRAK1. PLoS Pathog, 2013,9: e1003248.
[17] Yang Q, Fu S, Wang J. Hepatitis C virus infection decreases the expression of Toll-like receptors 3 and 7 via upregulation of miR-758. Arch Virol, 2014, 159: 2997-3003.
[18] Yoshikawa T, Takata A, Otsuka M, et al. Silencing of microRNA-122 enhances interferon-α signaling in the liver through regulating SOCS3 promoter methylation. Sci Rep, 2012, 2 :637.
[19] Xu G, Yang F, Ding CL, et al. MiR-221 accentuates IFN s anti-HCV effect by downregulating SOCS1 and SOCS3. Virology, 2014, 462: 343-350.
[20] Murakami Y, Tanaka M, Toyoda H, et al. Hepatic microRNA expression is associated with the response to interferon treatment of chronic hepatitis C. BMC Med Genomics, 2010, 3: 48.
[21] Estrabaud E, Lapalus M, Brot P, et al. Reduction of MicroRNA 122 Expression in IFNL3 CT/TT Carriers and during Progression of Fibrosis in Patients with Chronic Hepatitis C. J Virol,2014;88:6394-6402.
[22] McFarland AP, Horner SM, Jarret A, et al. The favorable IFNL3 genotype escapes mRNA decay mediated by AU-rich elements and hepatitis C virus-induced microRNAs. Nat Immunol. 2014;15:72-79.
[23] Jiang M, Broering R, Trippler M, et al. MicroRNA-155 controls Toll-like receptor 3-and hepatitis C virus-induced immune responses in the liver. J Viral Hepat, 2014, 21: 99-110.
[24] Choi Y, Dienes HP, Krawczynski K. Kinetics of miR-122 expression in the liver during acute HCV infection. PloS one, 2013, 8: e76501.
[25] Hou W, Bukong TN, Kodys K, et al. Alcohol facilitates HCV RNA replication via upregulation of miR-122 expression and inhibition of cyclin G1 in human hepatoma cells. Alcohol Clin Exp Res, 2013,37: 599-608.
[26] Fehr C, Conrad KD, Niepmann M. Differential stimulation of hepatitis C virus RNA translation by microRNA-122 in different cell cycle phases. Cell Cycle, 2012, 11: 277-285.
[27] Hou W, Tian Q, Zheng J, et al. MicroRNA-196 represses Bach1 protein and hepatitis C virus gene expression in human hepatoma cells expressing hepatitis C viral proteins. Hepatology, 2010, 51: 1494-1504.
[28] Liu X, Wang T, Wakita T, et al. Systematic identification of microRNA and messenger RNA profiles in hepatitis C virus-infected human hepatoma cells. Virology, 2010, 398: 57-67.
[29] Marquez RT, Bandyopadhyay S, Wendlandt EB, et al. Correlation between microRNA expression levels and clinical parameters associated with chronic hepatitis C viral infection in humans. Lab Invest, 2010,90: 1727-1736.
[30] Bandyopadhyay S, Friedman RC, Marquez RT, et al. Hepatitis C virus infection and hepatic stellate cell activation downregulate miR-29: miR-29 overexpression reduces hepatitis C viral abundance in culture. J Infect Dis, 2011, 203: 1753-1762.
[31] Ramachandran S, Ilias Basha H, Sarma NJ, et al. Hepatitis C virus induced miR200c down modulates FAP-1, a negative regulator of Src signaling and promotes hepatic fibrosis. PLoS One, 2013, 8: e70744.
[32] Sarma NJ, Tiriveedhi V, Crippin JS, et al. Hepatitis C virus-induced changes in microRNA 107 (miRNA-107) and miRNA-449a modulate CCL2 by targeting the interleukin-6 receptor complex in hepatitis. J Virol, 2014, 88: 3733-3743.
[33] Zhang Y, Wei W, Cheng N, et al. Hepatitis C virus-induced up-regulation of microRNA-155 promotes hepatocarcinogenesis by activating Wnt signaling. Hepatology, 2012, 56: 1631-1640.
[34] Varnholt H, Drebber U, Schulze F, et al. MicroRNA gene expression profile of hepatitis C virus-associated hepatocellular carcinoma.Hepatology, 2008,47:1223-1232.
[35] Xu H, Li G, Yue Z, et al. HCV core protein-induced upregulation of microRNA-196a promotes aberrant proliferation in hepatocellular carcinoma by targeting FOXO1. Mol Med Rep. 2016,13: 5223-5229. |
